How do blood clots form? Hyperlipidemia patients, do blood clots automatically grow in blood vessels?
A thrombus is a disease that can appear in any artery, including veins. The murderer of many cardiovascular diseases is actually thrombosis, which includes the well-known cerebral infarction and myocardial infarction. According to a statistics, cardiovascular and cerebrovascular diseases are the number one cause of death of Chinese residents due to diseases.
Moreover, the most terrifying thing about thrombus is that it is hidden in the blood vessels, which can neither be seen with the naked eye nor felt its existence! When it comes to the causes of thrombosis, people may first think of high blood lipids and high fat diets. However, is thrombus formation really that simple?
Hyperlipidemia is indeed a major risk factor for inducing thrombosis, but this is not absolute! Because thrombosis is not only the result of lipid accumulation and blood viscosity. From a scientific point of view, thrombosis is actually caused by the excessive aggregation of thrombocyte "platelets" in the blood.
Therefore, if you want to know why thrombus is formed, you must first know why platelets aggregate!
Under normal circumstances, platelets play a role in blood coagulation in the human body. For example, after trauma and bleeding on the body surface, this special signal will be immediately transmitted to the brain along with the nerves. It collects at the wound and forms a mass around the wound, blocking the capillaries and preventing them from continuing to bleed. The blood scab we see on the body surface is actually the product of platelet aggregation;
And if this happens on the blood vessels, for example, if the arterial blood vessels are damaged, the platelets will also aggregate here, but instead of forming a blood scab on the human body, it becomes a so-called "thrombus"!
In this case, is the thrombus in the cardiovascular and cerebrovascular vessels formed because of blood vessel damage?
In layman's terms, the formation of arterial thrombosis is indeed the product of blood vessel damage. But it's not that the blood vessel ruptures and bleeds, but the inner wall of the blood vessel is damaged! Under the influence of long-term "three-high" disease, the patient's arteries will gradually develop atherosclerosis, which will trigger an inflammatory reaction in the blood vessels, and lipids will continue to accumulate here to form plaques;
After the plaque ruptures, the deposited fat is exposed to the blood, which is equivalent to forming a nucleus that attracts platelets to aggregate. After receiving this signal, a large number of platelets will immediately integrate and move towards this place, and gather around to form thrombus!
Therefore, hyperlipidemia is not the direct murderer of thrombosis, it just makes the lipids in the blood exceed the standard, and fat does not condense into a group in the blood, and simple hyperlipidemia will not make blood vessels Thrombosis occurs directly. However, persistently elevated blood lipids can lead to atherosclerosis and plaque formation! When the plaque ruptures, a blood clot occurs.
In addition, it needs to be explained that the relationship between high blood lipids and blood viscosity!
Many people think that high blood lipids cause blood viscosity, but in fact blood viscosity is the murderer that induces high blood lipids. The so-called blood viscosity means that the red blood cells in the blood gather into piles and lose their original gaps and aggregations, or the red blood cells in the blood have a reduced ability to bend and deform when passing through tiny capillaries, resulting in an increase in blood viscosity. , After the blood viscosity rises, the circulation resistance increases, which leads to the accumulation of lipid substances in the body, which induces problems such as endogenous hyperlipidemia and hypertension.
Therefore, in order to prevent the formation of thrombosis, the most important thing is to remove the conditions for platelet aggregation, that is, to avoid arteriosclerosis and plaque formation. Especially for patients with high blood pressure and high blood lipids, treatment should be strictly in accordance with the doctor's recommendations. Only by controlling the primary disease can the risk of thrombosis be reduced.